Avoid high-nitrogen fertilizer because the plant will produce all foliage and no flowers. Use a water-soluble fertilizer with a balanced formula, such as 15-15-15, or a blooming formula, such as 7-9-5. Too much fertilizer can contribute to weak or leggy growth, so only supply fertilizer during the growing season and discontinue feeding in autumn and winter to give the plant a chance to rest. Fertilization Zigzag plants will grow well without fertilizer, but a boost of nutrients during the summer months will help encourage the plant to produce its showy red flower bracts, which appear in late spring and early summer.
Let the soil reach a state of visible dryness between each watering but don't let the plant wilt because it can cause damage to the stems. Be sure to pour out any excess water from the catch basin underneath the pot so it's not sitting in standing water. Add water to the soil until it trickles from the drainage holes at the bottom of the pot. Weekly watering is sometimes necessary during hot weather but always check the soil with the tip of a finger before watering and only water when it feels dry beneath the surface. Water deeply but infrequently during the summer months and then reduce watering in autumn and winter when the weather is cool. They need consistent moisture year-round, but too much moisture at any given time can cause root rot, which is perhaps the most significant health risk for succulents. Watering The right watering technique is key to successfully caring for zigzag plants. It can thrive indoors at room temperature if it’s leaves and branches are misted weekly. While this tropical plant does not need humidity to survive, for optimum growth it’s best to plant in a combination of sunlight and moisture. It can thrive indoors at room temperature if kept it in a humid area. It’s a tropical plant needing a combination of sunlight and moisture. Temperature If temperatures get below 40° degrees Fahrenheit during the winter, move the plant indoors. Overly bright sunlight may scorch the tender foliage, so keep an eye on it and move it or create some shade if this happens. If you position it in a very sunny window, a lightweight curtain or slatted blinds turned to half-position works to give this plan the sun exposure it needs. The profile of steady‐state pathogen levels resembles the profile of the expository zigzag model.Light Zig-Zag plant does best with plenty of indirect sunlight. (c) Levels of callose and pathogen (arbitrary units) after 200 time units, when a steady state has been reached, demonstrating the influence of PTI, ETI and effector action with respect to the absence of a host immune response. (b) Levels of callose and pathogen (arbitrary units) over 200 time units of simulation, for systems where the host shows: no resistance response: PTI, host shows PTI only PTI+ETS, host shows PTI only but the pathogen suppresses PTI by effector production PTI+ETS+ ETI, host exhibits PTI and ETI, but the pathogen suppresses PTI by effector production. (b), (c) Quantitative output of the immune system model. Activated R protein* also increases the rate at which the local microbial population is depleted, as an abstraction of ETI. As a proxy for PTI, callose also increases the rate at which local microbe populations decline, and acts to reduce the rate at which effector is translocated into the cell.
Within the cell compartment, if there is activated PRR*, the plant also produces callose, as a proxy for PTI activity. The effector may be internalized to the cell (translocation), where it may interact reversibly with plant R protein to produce an activated R protein* species. The PAMP may interact reversibly with plant pattern‐recognition receptor ( PRR) to produce an activated PRR* species. by diffusion or destruction) in the extracellular compartment. While local to the plant, the microbe produces two species: pathogen‐associated molecular patterns ( PAMP) and effector both species can be lost (e.g. The rate at which the microbe is ‘destroyed’, which may be interpreted, for example, as microbial movement or death, is enhanced by pattern‐triggered immunity ( PTI) and effector‐triggered immunity ( ETI). In the extracellular compartment the local microbial population is drawn from a remote bulk population and is also ‘destroyed’, as indicated by the arrow pointing to the empty set symbol (ϕ).
The system is divided into two ‘compartments’: extracellular (external to the cell wall) and intracellular (internal to the cell wall). (a) Schematic diagram of the model plant immune system (BioModels: MODEL1408280000).
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